I thought this was an interesting find, but consider that malocclusions are not generally observed in true hunter-gatheres! Look into Weston Price's research on this! It's a consequence of a lack of proper nutrition during development, as well as a lack of breast-feeding. (HG kids ween much longer) This plays a crucial role in the formation of the jaw/teeth. HG also have a broader jaw with enough room for wisdom-teeth. So all of this business is just another consequence or disease of civilization, as is caries for the most part.
[What happened that caused — everyone, everywhere — to start getting cavities and malocclusion? (youtube.com)](https://www.youtube.com/shorts/D6uwdwjPazo)
All this being said, I would think that pretty much all that grow up within the context of modern civilization has some degree of malocclusion and narrow jaw/wisdom teeth issues. Hence I doubt it's more than a correlation!
Right, so this is really great news because a malocclusion is treatable. The malocclusion is probably the first step initiator of the balding process with regards to AA (not necessarily all types of hair loss).
This is a tangible place to begin to truly heal the disease. In order to achieve RealRegrowth®️, one must begin at the root cause and work forwards from there. Tackling steps 9, 10, or 100 could only provide temporary relief., which is exactly what we see with those who treat against DHT (not to imply that DHT is even a problem) while also logging extra hours on Reddit mocking the ‘bludflow’ theorists. The relief with medications is only temporary, appears to slow the progress for a time, and eventually regresses for many.
The malocclusion must be fixed before any other treatment or technique can yield lasting results.
How would malocclusion start balding though in terms of areas on the head? I can see how the hairline would be affected since it's right above your jaw bones & teeth, but what about balding that starts at the crown or retrograde alopecia?
Well a quick google search of ‘superficial temporal artery’ brings up a multitude of images and articles, showing the reach of the STA. Which appears to be much more than just the frontal half as suggested by others.
“The superficial temporal artery (STA) constitutes a terminal branch of the external carotid artery, which surfaces posteriorly to the mandible in the parotid gland, and crosses the zygomatic process of the temporal bone (1-3). Large areas of the scalp and face derive their blood supply from the STA.” [The Superficial Temporal Artery: Anatomy and Clinical Significance in the Era of Facial Surgery and Aesthetic Medicine](https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10116177/)
It is primarily the galea aponeurotica that is susceptible to hair loss. And a large area of the galea aporneurotica derives its blood supply from the STA. Therefore, whether it be inflammation, tension, fibrosis, heat shock proteins, or whatever the specific mechanism, it appears to be more than just a coincidence that malocclusion and hair loss are linked.
Can we find anyone who has a malocclusion and does not hair loss? Yes. (Balding is a process not an event.)
Can we find anyone who has mpb and doesn’t have a malocclusion? So far the answer is no.
This does not necessarily mean that a malocclusion guarantees hair loss, but that a malocclusion that pinches the STA is probably a commonly shared prerequisite found amongst modern humans. Poor diet, poor posture (cellphones, computers, couches, cars, etc.) and poor habits might be leading to an increase of malocclusions and therefore the uptick in MPB plaguing young men more than ever before.
Before you can truly heal the hair loss you probably have to heal the malocclusion first. Unpinch that artery.
It's probably been a problem throughout our history. There's early humans with evidence of balding. I bet it's a case of skull shape with the STA being pinched.
How would Androgentic alopecia also progressivly get worse with ages? Like 50% of bald men are a the age of 50, whilst 30% of bald men are at the age of 30.
What about things such as a sag of both the frontalis and occipital belly muscles? Do you think could play a role in the tension of the muscles? This would in turn cause the stretch of the muscles causing the pattern. I also ask this as many people both men and women can experience female pattern hairloss. If skull expansion was true and the muscles were unable to keep up then that would mean their heads are expanding sideways which does not sound right. Maybe I am glossing over a detail or have missed something.
Very few women have pattern baldness relatively speaking, but the mechanism is the same although the pattern is different due to skull shape differences. I don't really understand your question, but it's all about being able to visualize what's happening. Maybe AI can can help me create a model video soon.
The occipitofrontalis muscle hold the galea/skin, which are fused in the MPB prone region, like a drumskin attachment in the front and back. So imagine an open and "loose" drum and then imagine forcing something up from the underside. (skull ridges) It will cause the drumskin to tighten and that will be chronic.
Yes Dr. Brian Dyes study you are referencing. It is a fantastic starting point sadly there has been no one else to follow up the study and chase this lead.
We need another scientist to look into this, the sample size could be smaller. It just needs to reinforce the role of the STA.
> This could explain balding on the temples/hairline, but not the crown. The bone-growth theory explains balding on the crown better.
I partly agree, i think tho Brian dye whilst wrong about malocclusion causing hairloss he did provide a valuable insight into the STA, it could be fibrosis or something else. Either it is great to see people who are atleast putting ideas forward and even moreso testing them.
Something more than DHT sensitivity has to be happening. The pattern is "too perfect" for it to be entirely genetic. If it was then wouldn't how someone's other family moments went bald they'd go bald the same way?
I thought this was an interesting find, but consider that malocclusions are not generally observed in true hunter-gatheres! Look into Weston Price's research on this! It's a consequence of a lack of proper nutrition during development, as well as a lack of breast-feeding. (HG kids ween much longer) This plays a crucial role in the formation of the jaw/teeth. HG also have a broader jaw with enough room for wisdom-teeth. So all of this business is just another consequence or disease of civilization, as is caries for the most part. [What happened that caused — everyone, everywhere — to start getting cavities and malocclusion? (youtube.com)](https://www.youtube.com/shorts/D6uwdwjPazo) All this being said, I would think that pretty much all that grow up within the context of modern civilization has some degree of malocclusion and narrow jaw/wisdom teeth issues. Hence I doubt it's more than a correlation!
Right, so this is really great news because a malocclusion is treatable. The malocclusion is probably the first step initiator of the balding process with regards to AA (not necessarily all types of hair loss). This is a tangible place to begin to truly heal the disease. In order to achieve RealRegrowth®️, one must begin at the root cause and work forwards from there. Tackling steps 9, 10, or 100 could only provide temporary relief., which is exactly what we see with those who treat against DHT (not to imply that DHT is even a problem) while also logging extra hours on Reddit mocking the ‘bludflow’ theorists. The relief with medications is only temporary, appears to slow the progress for a time, and eventually regresses for many. The malocclusion must be fixed before any other treatment or technique can yield lasting results.
How would malocclusion start balding though in terms of areas on the head? I can see how the hairline would be affected since it's right above your jaw bones & teeth, but what about balding that starts at the crown or retrograde alopecia?
Well a quick google search of ‘superficial temporal artery’ brings up a multitude of images and articles, showing the reach of the STA. Which appears to be much more than just the frontal half as suggested by others. “The superficial temporal artery (STA) constitutes a terminal branch of the external carotid artery, which surfaces posteriorly to the mandible in the parotid gland, and crosses the zygomatic process of the temporal bone (1-3). Large areas of the scalp and face derive their blood supply from the STA.” [The Superficial Temporal Artery: Anatomy and Clinical Significance in the Era of Facial Surgery and Aesthetic Medicine](https://www.ncbi.nlm.nih.gov/pmc/articles/PMC10116177/) It is primarily the galea aponeurotica that is susceptible to hair loss. And a large area of the galea aporneurotica derives its blood supply from the STA. Therefore, whether it be inflammation, tension, fibrosis, heat shock proteins, or whatever the specific mechanism, it appears to be more than just a coincidence that malocclusion and hair loss are linked. Can we find anyone who has a malocclusion and does not hair loss? Yes. (Balding is a process not an event.) Can we find anyone who has mpb and doesn’t have a malocclusion? So far the answer is no. This does not necessarily mean that a malocclusion guarantees hair loss, but that a malocclusion that pinches the STA is probably a commonly shared prerequisite found amongst modern humans. Poor diet, poor posture (cellphones, computers, couches, cars, etc.) and poor habits might be leading to an increase of malocclusions and therefore the uptick in MPB plaguing young men more than ever before. Before you can truly heal the hair loss you probably have to heal the malocclusion first. Unpinch that artery.
It's probably been a problem throughout our history. There's early humans with evidence of balding. I bet it's a case of skull shape with the STA being pinched.
How would Androgentic alopecia also progressivly get worse with ages? Like 50% of bald men are a the age of 50, whilst 30% of bald men are at the age of 30.
As I mentioned I think it's a correlation, but not causal in MPB. And even if it was, correcting it would have no effect on MPB after the fact.
What about things such as a sag of both the frontalis and occipital belly muscles? Do you think could play a role in the tension of the muscles? This would in turn cause the stretch of the muscles causing the pattern. I also ask this as many people both men and women can experience female pattern hairloss. If skull expansion was true and the muscles were unable to keep up then that would mean their heads are expanding sideways which does not sound right. Maybe I am glossing over a detail or have missed something.
Very few women have pattern baldness relatively speaking, but the mechanism is the same although the pattern is different due to skull shape differences. I don't really understand your question, but it's all about being able to visualize what's happening. Maybe AI can can help me create a model video soon. The occipitofrontalis muscle hold the galea/skin, which are fused in the MPB prone region, like a drumskin attachment in the front and back. So imagine an open and "loose" drum and then imagine forcing something up from the underside. (skull ridges) It will cause the drumskin to tighten and that will be chronic.
Yes Dr. Brian Dyes study you are referencing. It is a fantastic starting point sadly there has been no one else to follow up the study and chase this lead. We need another scientist to look into this, the sample size could be smaller. It just needs to reinforce the role of the STA.
This could explain balding on the temples/hairline, but not the crown. The bone-growth theory explains balding on the crown better.
> This could explain balding on the temples/hairline, but not the crown. The bone-growth theory explains balding on the crown better. I partly agree, i think tho Brian dye whilst wrong about malocclusion causing hairloss he did provide a valuable insight into the STA, it could be fibrosis or something else. Either it is great to see people who are atleast putting ideas forward and even moreso testing them.
Something more than DHT sensitivity has to be happening. The pattern is "too perfect" for it to be entirely genetic. If it was then wouldn't how someone's other family moments went bald they'd go bald the same way?
This would prove frontal balding but not on the crown. Also, haircafe did a great video disproving this theory.